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JODDD now with visual abstracts. Authors can now add a visual abstract to their submission.

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*This Platinum Open Access journal publishes articles totally free of charge for the authors and provides unrestricted access to the published content through its website and open access repositories such as PubMed Central.

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J Dent Res Dent Clin Dent Prospects. 2018;12(3): 159-165. doi: 10.15171/joddd.2018.025
PMID: 30443300        PMCID: PMC6231148

Original Article

Evaluation of the effect of IL-36γ expression on chronic periodontitis by enhancing the MAPK and TLR4 signaling pathways: A basic research

Amir Reza Babaloo 1, Adileh Shirmohammadi 1,2, Siamak Sandoghchian shotorbani 3, Ashkan Kamalzadeh 4 * , Shima Ghasemi 5

Cited by CrossRef: 4

1- Korkmaz H, Hatipoğlu M, Kayar N. Interleukin‐38: A crucial player in periodontitis. Oral Diseases. 2024;30(4):2523 [Crossref]
2- Tongmuang N, Cai K, An J, Novy M, Jensen L. Floxed Il1rl2 Locus with mCherry Reporter Element Reveals Distinct Expression Patterns of the IL-36 Receptor in Barrier Tissues. Cells. 2024;13(9):787 [Crossref]
3- Saito K, Iwata Y, Fukushima H, Watanabe S, Tanaka Y, Hasegawa Y, Akiyama M, Sugiura K. IL-36 receptor antagonist deficiency resulted in delayed wound healing due to excessive recruitment of immune cells. Sci Rep. 2020;10(1) [Crossref]
4- Hinson A, Hawkes C, Blake C, Fitzsimonds Z, Zhu B, Buck G, Lamont R, Miller D, Monack D. Treponema denticola Induces Interleukin-36γ Expression in Human Oral Gingival Keratinocytes via the Parallel Activation of NF-κB and Mitogen-Activated Protein Kinase Pathways. Infect Immun. 2022;90(10) [Crossref]

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