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JODDD now with visual abstracts. Authors can now add a visual abstract to their submission.

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J Dent Res Dent Clin Dent Prospects. 2018;12(3): 159-165. doi: 10.15171/joddd.2018.025
PMID: 30443300        PMCID: PMC6231148

Original Article

Evaluation of the effect of IL-36γ expression on chronic periodontitis by enhancing the MAPK and TLR4 signaling pathways: A basic research

Amir Reza Babaloo 1, Adileh Shirmohammadi 1,2, Siamak Sandoghchian shotorbani 3, Ashkan Kamalzadeh 4 * , Shima Ghasemi 5

Cited by CrossRef: 3


1- Korkmaz H, Hatipoğlu M, Kayar N. Interleukin‐38: A crucial player in periodontitis. Oral Diseases. 2023; [Crossref]
2- Saito K, Iwata Y, Fukushima H, Watanabe S, Tanaka Y, Hasegawa Y, Akiyama M, Sugiura K. IL-36 receptor antagonist deficiency resulted in delayed wound healing due to excessive recruitment of immune cells. Sci Rep. 2020;10(1) [Crossref]
3- Hinson A, Hawkes C, Blake C, Fitzsimonds Z, Zhu B, Buck G, Lamont R, Miller D, Monack D. Treponema denticola Induces Interleukin-36γ Expression in Human Oral Gingival Keratinocytes via the Parallel Activation of NF-κB and Mitogen-Activated Protein Kinase Pathways. Infect Immun. 2022;90(10) [Crossref]